Samuel D. Chauvin

2 months ago | cell.com | Subhajit Poddar |Samuel D. Chauvin |Christopher Archer |Wei Qian |Jean A. Castillo-Badillo |Xin Yin | +25 more

1Division of Rheumatology, Department of Medicine, University of Pennsylvania Perelman School of Medicine, Philadelphia, PA 19104, USA 2Department of Cell Biology and Physiology, Washington University School of Medicine, St Louis, MO 63110, USA 3Department of Medicine, Washington University School of Medicine, St Louis, MO 63110, USA 4Immunity and Pathogenesis Program, Sanford Burnham Prebys Medical Discovery Institute, La Jolla, CA 92037, USA 5State Key Laboratory of Veterinary...

Feb 12, 2024 | nature.com | Ann Park |Justin Q. Gabrielski |Hyoungjun Ham |Jean Michel Chanchu |Aaron R. Morawski |Aiman J. Faruqi | +14 more

Correction to: Nature Immunology https://doi.org/10.1038/s41590-023-01691-y, published online 3 January 2024. In the version of the article initially published, Buket Dalgıç (Department of Pediatric Gastroenterology, Gazi University Faculty of Medicine, Ankara, Turkey) was mistakenly omitted from the author list and has now been added in the HTML and PDF versions of the article. About this articlePark, A.Y., Leney-Greene, M., Lynberg, M. et al.

Jan 3, 2024 | nature.com | Ann Park |Aiman J. Faruqi |Samuel D. Chauvin |Holden T Maecker |Altan Kara |Odile Filhol | +14 more

AbstractPreserving cells in a functional, non-senescent state is a major goal for extending human healthspans. Model organisms reveal that longevity and senescence are genetically controlled, but how genes control longevity in different mammalian tissues is unknown.