Articles

  • 1 week ago | cen.acs.org | Sarah Braner

    With $90 million in funding, Syndeio Biosciences has launched to target synaptopathies. These diseases and disorders, such as Alzheimer's disease and major depressive disorder, are thought to result from damaged neural networks. In addition to cash, the company has several assets in hand, including a lead drug candidate, zelquistinel, that is currently in Phase 2 clinical trials to treat major depressive disorder.

  • 1 month ago | cen.acs.org | Sarah Braner

    Researchers at Vanderbilt University have found that boosting a signaling pathway in the brain can prolong ketamine's antidepressant effects, at least in mice. The team found that compared with mice that received only ketamine, mice who got the modified treatment showed less-pronounced depressive behaviors 8 weeks after the treatment ( Science 2025, DOI: 10.1126/science.abb6748).

  • 1 month ago | cen.acs.org | Sarah Braner

    A new drug delivery method could improve treatments for inflammatory bowel disease (IBD) like ulcerative colitis and Crohn's disease. Currently available oral drugs for IBD must transit the entire upper digestive tract before they get to the lower gastrointestinal (GI) tract-the area where they're actually needed, says Laura Sly, a professor in the pediatrics department at the University of British Columbia (UBC).

  • 1 month ago | cen.acs.org | Sarah Braner

    Grove Biopharma has raised a $30 million series A round to develop Bionic Biologics, also known as protein-like polymers (PLP), which were invented by Nathan Gianneschi at Northwestern University. A PLP is a polymer of peptides that mimics a protein but is more stable and more cell permeable. It is composed of a hydrophobic, semiflexible backbone and hydrophilic ends. That structure causes the PLP to fold in on itself in an aqueous solution, mimicking a globular protein.

  • 1 month ago | cen.acs.org | Sarah Braner

    As the field of proteomics continues to advance, scientists are using new methods to answer longstanding questions about rare diseases. One such disease is α-1-antitrypsin deficiency (AATD), a proteotoxic liver disease, in which misfolded and unfolded proteins cause cell death. In this case, a deletion in the gene for α-1-antitrypsin leads to a misfolded mutant, which accumulates and eventually results in cell death.

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