Articles
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1 day ago |
jci.org | Sarah Costantino |Shafeeq A. Mohammed |Francesco Paneni |Aida Collado
AbstractEndothelial dysfunction remains a cornerstone of diabetic vascular complications. RBCs emerge as pivotal players in endothelial dysfunction, yet the underlying mechanisms remain elusive. In this issue of the JCI, Collado et al. show that the detrimental action of RBCs on the endothelium is mediated by extracellular vesicles (EVs).
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