Wei Shuai

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USP38 Exacerbates Atrial Inflammation, Fibrosis, and Susceptibility to Atrial Fibrillation After Myocardial Infarction in Mice

Aug 6, 2024 | digitalcommons.library.tmc.edu | Tingting Yu |Wei Shuai |Tao Chen |Jingjing Zhang

BACKGROUND: Inflammation plays an important role in the pathogenesis of atrial fibrillation (AF) after myocardial infarction (MI). The role of USP38, a member of the ubiquitin-specific protease family, on MI-induced atrial inflammation, fibrosis, and associated AF is unclear.

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