Xiaoyan Xu

2 months ago | cell.com | Jun Xiao |Weiyun Li |Xin Zheng |Linlin Qi |Hui Wang |Chi Zhang | +16 more

Main text(Immunity 52, 109–122.e1–e6; January 14, 2020)The authors note that Figure S6C showing normal lung tissue in the Akt3+/- Mock group is partially overlapped with the Akt3+/+ Mock control group. Figure S6A representing normal lung tissue in the AY9944-PBS group was mistakenly uploaded with one image from the PBS control group that also represents normal lung tissue. We have now provided the correct images for the Akt3+/- Mock (Figure S6C) and AY9944-PBS group (Figure S6A).

Mar 13, 2025 | biorxiv.org | Jun Lyu |Xiaoyan Xu |Chongyi Chen

AbstractSequencing newly synthesized transcriptome alongside regular transcriptome in single cells enables the study of gene expression temporal dynamics during rapid chromatin and gene regulation processes. Existing assays for profiling single-cell newly synthesized transcriptome often require specialized technical expertise to achieve high cellular throughput, limiting their accessibility.

Aug 23, 2024 | biorxiv.org | Jun Lyu |Xiaoyan Xu |Chongyi Chen

AbstractSequencing newly synthesized transcriptome alongside regular transcriptome in single cells enables the study of gene expression temporal dynamics during rapid chromatin and gene regulation processes. However, existing assays to profile single-cell newly synthesized transcriptome require in-house technical expertise to achieve high cellular throughput, limiting their widespread application.

Aug 1, 2024 | mdpi.com | Xiaoyan Xu |Hong Zhao

This is an early access version, the complete PDF, HTML, and XML versions will be available soon. Open AccessArticle by Xiaoyan Xu SciProfiles Scilit Preprints.org Google Scholar and Hong Zhao SciProfiles Scilit Preprints.org Google Scholar * School of Economics and Management, University of Chinese Academy of Sciences, Beijing 100190, China *Author to whom correspondence should be addressed.

Jan 11, 2024 | pubs.rsc.org | Junyi Li |Fei Peng |Hui Huang |Xiaoyan Xu

Characterization, mechanism and in vivo validation of Helicobacter pylori antagonism by probiotics screened from infants’ feces and oral cavity† Helicobacter pylori (H. pylori) infection is a major cause of chronic gastritis, intestinal metaplasia, and gastric carcinoma. Antibiotics, the conventional regimen for eliminating H. pylori, cause severe bacterial resistance, gut dysbiosis and hepatic insufficiency.