Allison M Dyevoich

Jun 27, 2024 | science.org | Krishnan K Mahadevan |Dawid Zyla |Allison M Dyevoich |Yang Chen

1F. Balkwill, A. Mantovani, Inflammation and cancer: Back to Virchow?Lancet 357, 539–545 (2001). 2I. Gukovsky, N. Li, J. Todoric, A. Gukovskaya, M. Karin, Inflammation, autophagy, and obesity: Common features in the pathogenesis of pancreatitis and pancreatic cancer. Gastroenterology 144, 1199–209.e4 (2013). 3M. Perez-Moreno, W. Song, H. A. Pasolli, S. E. Williams, E. Fuchs, Loss of p120 catenin and links to mitotic alterations, inflammation, and skin cancer. Proc. Natl. Acad. Sci. U.S.A. 105, 15399–15404 (2008). 4A.

Jun 27, 2024 | science.org | Krishnan K Mahadevan |Dawid Zyla |Allison M Dyevoich |Yang Chen

Editor’s summaryInflammation of the pancreas, known as pancreatitis, can influence the emergence and progression of pancreatic cancer. How the body’s immune system responds to pancreatitis versus pancreatic cancer remains unclear. Mahadevan et al. report that pancreatitis leads to the recruitment of a class of immune cells called conventional dendritic cells that can help to protect tissue from self-destruction by T lymphocytes.

Mar 6, 2023 | biorxiv.org | Krishnan K Mahadevan |Allison M Dyevoich |Yang Chen |Bingrui Li

Thank you for your interest in spreading the word about bioRxiv. NOTE: Your email address is requested solely to identify you as the sender of this article. Your Email * Your Name * Send To * Enter multiple addresses on separate lines or separate them with commas. Message Subject (Your Name) has forwarded a page to you from bioRxiv Message Body (Your Name) thought you would like to see this page from the bioRxiv website.