Humanitas Clinical

Jul 8, 2024 | link.springer.com | Maria Rosaria |Humanitas Clinical

Correction: J Anesth Analg Crit Care 4, 42 (2024)https://doi.org/10.1186/s44158-024-00161-7In the original publication of this article [1] there was an incorrect author name. The incorrect and correct information is listed in this correction article. The original article has been updated.

May 1, 2024 | trialbulletin.com | Humanitas Clinical

OverviewFluids are considered the primary treatment for critically ill patients admitted to the intensive care unit (ICU), aiming to replace losses and or to enhance venous return, stroke volume, and consequently, cardiac output and tissue oxygen delivery. The modalities, volumes, and targets employed to titrate fluid therapy vary significantly in current clinical practice, as shown by the original FENICE study 10 years ago. FENICE studied how fluid challenges are given at the bedside.

Apr 29, 2024 | academic.oup.com | Di Lauro |Humanitas Clinical

Accepted manuscripts are PDF versions of the author’s final manuscript, as accepted for publication by the journal but prior to copyediting or typesetting. They can be cited using the author(s), article title, journal title, year of online publication, and DOI. They will be replaced by the final typeset articles, which may therefore contain changes. The DOI will remain the same throughout. This content is only available as a PDF. © The Author(s) 2024.

Apr 22, 2024 | europeanurology.com | Giuseppe Basile |Andrea Gallioli |Humanitas Clinical |Anthony Gallagher |Pietro Diana |Alessandro Larcher | +9 more

AbstractDifferent training programs have been developed to improve trainee outcomes in urology. However, evidence on the optimal training methodology is sparse. Our aim was to provide a comprehensive description of the training programs available for urological robotic surgery and endourology, assess their validity, and highlight the fundamental elements of future training pathways. We systematically reviewed the literature using PubMed/Medline, Embase, and Web of Science databases.

Jan 18, 2024 | academic.oup.com | Humanitas Clinical

DNAJC6 encodes auxilin, a co-chaperone protein involved in clathrin-mediated endocytosis (CME) at the presynaptic terminal. Biallelic mutations in DNAJC6 cause a complex, early-onset neurodegenerative disorder characterized by rapidly progressive parkinsonism-dystonia in childhood. The disease is commonly associated with additional neurodevelopmental, neurological and neuropsychiatric features.