Will Flanigan

Oct 23, 2024 | nature.com | Michael Alexanian |Tomohiro Nishino |Lin Ye |Clara Lee |Will Flanigan |Zachary Gardner | +7 more

AbstractChronic inflammation and tissue fibrosis are common responses that worsen organ function, yet the molecular mechanisms governing their cross-talk are poorly understood. In diseased organs, stress-induced gene expression changes fuel maladaptive cell state transitions1 and pathological interaction between cellular compartments.