Journal of Cell Biology
The Journal of Cell Biology (JCB) is a globally recognized, peer-reviewed publication that is managed by The Rockefeller University and distributed by Rockefeller University Press.
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Jun 11, 2024 |
rupress.org | Lausanne Branch |Johanna Chiffelle |Alexandre Harari
In cancer research, antigens have long been recognized as pivotal triggers of immune responses and potential therapeutic targets. Antigens are commonly categorized into tumor-associated antigens (TAAs) or canonical neoantigens (Peri et al., 2023). TAAs are self-antigens, showing varied expression patterns within tumors.
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Jun 4, 2024 |
rupress.org | Heidi Ulrichs |Shashank Shekhar
Actin and microtubule dynamics are essential for key cellular processes such as cell migration, cell division, and transport of organelles or vesicles. Although the activities of these two distinct cytoskeletal networks are often studied one at a time, the idea that their activities are intertwined and that they influence each other’s dynamics has been around for over four decades (1).
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Jun 3, 2024 |
rupress.org | Richard John |Sophie Gray |Cecile Fort |Richard Wheeler
Subjects: Cilia, Microbiology, Technology Motile eukaryotic flagella and cilia have a complex architecture. They are assembled, maintained, and function through the action of two types of motor proteins.
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May 6, 2024 |
rupress.org | Melanie Krause |Jerzy Samolej |Artur Yakimovich
Cell autonomous immunity represents the first line of defense used by cells to combat incoming pathogens (Randow et al., 2013). Among the various strategies employed for detection and elimination of microbial invaders is xenophagy (Levine, 2005; Nakagawa et al., 2004a; Ohsumi, 2014).
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Apr 23, 2024 |
rupress.org | András N. Spaan
Subjects: Human disease genetics, Immunodeficiency, Innate immunity and inflammation Autoinflammation is the term used to describe disorders of hyperactivation of the immune system in the absence of autoimmunity. Around 50 monogenic inborn errors of immunity are known to underlie autoinflammatory syndromes. In an apparent paradox, autoinflammation can be associated with immunodeficiency. This paradox is illustrated by human deficiencies of the linear ubiquitin homeostasis.
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